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Angiogenesis occurs with contribution from VEGF and its receptors, eNOS and EGR-1, all of which have been shown to be affected and upregulated by BPC-157 [8, 38, 39, 70–72]

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Once a portion of the original H 2 O 2 substrate has been consumed by GPx, any remaining H 2 O 2 reacts with Fe 2+ ions from the FAS via a Fenton-type redox mechanism, oxidizing Fe 2+ to Fe³ +